Xu Lin

UFMylation of SHBs increases protein stability by antagonizing ubiquitination and promotes virion production

ABSTRACT Post-translational modifications orchestrate virus–host interactions, yet their roles in the hepatitis B virus (HBV) life cycle remain incompletely defined. The small hepatitis B surface antigen (SHBs)—the most abundant HBV protein—drives immune evasion, pathogenesis, an...