Siwen Li

Ferroptosis as a central mechanism in inhaled tris(1-chloro-iso-propyl) phosphate (TCIPP)-induced lung injury: Associations with pyroptosis and mitochondrial dynamic imbalance

), along with elevated ferroptosis markers (p53, Transferrin Receptor 1 (TFR1)) and reduced FTH1 levels following TCIPP inhalation. These effects were counteracted by the ferroptosis inhibitor Fer‑1 (0.8 mg/kg), which also attenuated pathological damage, ROS accumulation, and NLR...