姚凤良

Knockdown of CHI3L1 Attenuates Venous Intimal Hyperplasia by Modulating Vascular Inflammation and Oxidative Stress via the JNK/ERK Pathway

Background: Venous intimal hyperplasia (VIH) is the key pathological basis for early failure of arteriovenous fistula (AVF) and for stenosis of hemodialysis access. Chitinase-3-like protein 1 (CHI3L1) is highly expressed during cardiovascular remodeling and inflammation, but its...