Huan Liang

FTO-mediated m6A modification suppresses acute pancreatitis by downregulating 1,2-dilinoleoyl-GPC (18:2/18:2): An integrated multi-omics mechanistic study.

The pathophysiology of acute pancreatitis (AP), a common clinical emergency, is poorly understood. Previous studies have implicated N6-methyladenosine (m6A) modification in the pathogenesis of AP; however, the precise molecular mechanisms remain unclear. Whether metabolites parti...