Cornelius Deuschl

The kallikrein-kinin system modulates CD4+ T cell function and contributes to T cell migration into the brain after stroke

Abstract Ischemic stroke elicits a sustained thromboinflammatory response that critically shapes secondary brain injury. The kallikrein-kinin system (KKS) has been identified as the interface of vascular injury and inflammatory processes; however, its role in adaptive immunity re...